In the registrational Chinese phase-3, the GLP-1/glucagon dual agonist mazdutide produced 11-14% weight loss with low (~1%) discontinuation.
Mazdutide
MARKETEDGLP-1 and glucagon receptor dual agonist
Mazdutide has a current marketed status in the scoped regulatory record below. Results describe observations and do not provide treatment advice.
MARKETED China; Chronic weight management; Subcutaneous injection; as of 2026-07-13. Innovent reporting NMPA approval
The grade rates how strong the evidence is, not how good or safe the drug is. A low or very-low grade means the evidence is thin, not that an effect is harmful or ruled out.
Phase 3 GLORY-1 confirmed substantial weight loss vs placebo at 48 wk
High-dose mazdutide produced marked early weight loss vs placebo in Chinese overweight/obese adults
GLORY-1/GLORY-2 phase 3 (Chinese adults with obesity): mazdutide met key secondary endpoints including reduced waist circumference and liver-fat content; dual...
Mazdutide (IBI362; Innovent/Lilly, GLP-1/glucagon dual) has NO published dedicated human MASH histology trial. Human liver benefit is so far described within...
Mazdutide reduced systolic blood pressure vs placebo (GLORY-1)
Glucagon-receptor arm proposed to increase energy expenditure and improve hepatic fat metabolism (mammalian OXM analogue)
In a phase 3 trial in Chinese adults with T2D, both mazdutide doses were non-inferior and superior to dulaglutide 1.5 mg for HbA1c reduction at 28 weeks (head-to-head).
Reduced total cholesterol, triglycerides, LDL-C and serum uric acid
Mild-to-moderate GI TEAEs; no serious AEs in phase 1b
Registered or protocol records without results
These records remain discoverable, but they are not counted or presented as results.
Mazdutide (IBI362 / LY3305677; Innovent, licensed from Lilly) is a GLP-1/glucagon dual agonist (mammalian oxyntomodulin analogue) developed largely in Chinese...
DREAMS-3, the first head-to-head mazdutide-vs-semaglutide trial in Chinese adults with T2D + obesity, is designed but results pending (registry/protocol only).